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First published on July 20, 2004, doi:10.1177/0363546504263699
This version was published on September 1, 2004
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The American Journal of Sports Medicine 32:1492-1498 (2004)
© 2004 American Orthopaedic Society for Sports Medicine

Stability of Acromioclavicular Joint Reconstruction

Biomechanical Testing of Various Surgical Techniques in a Cadaveric Model

Ashwin V. Deshmukh, MD*,{dagger}, David R. Wilson, DPhil{ddagger}, Jeffrey L. Zilberfarb, MD§ and Gary S. Perlmutter, MD||

From the dagger; Department of Orthopaedics, Kaiser Permanente West Los Angeles Medical Center, Los Angeles, California, the {ddagger} Division of Orthopaedic Engineering Research, Department of Orthopaedics, University of British Columbia and Vancouver Coastal Health Research Institute, Vancouver, Canada, § Beth Israel Deaconess Medical Center, Boston, Massachusetts, and || Massachusetts General Hospital, Boston, Massachusetts

* Address correspondence to Ashwin V. Deshmukh, MD, Department of Orthopaedics, Kaiser Permanente West Los Angeles Medical Center, 6041 Cadillac Avenue, Los Angeles, CA 90034.

Background: Despite reports of excellent results with the Weaver-Dunn coracoacromial ligament transfer, many authors recommend augmenting the transfer with supplemental fixation. The authors of this study sought to determine whether there is a biomechanical basis for this assertion and which augmentative method, if any, most closely restored acromioclavicular motion to normal.

Hypothesis: Augmentative coracoclavicular fixation provides better restoration of normal acromioclavicular joint laxity and an increased failure load when compared with the Weaver-Dunn reconstruction alone.

Study Design: Controlled laboratory cadaveric study.

Methods: Native acromioclavicular joint motion was measured using an infrared optical measurement system. Acromioclavicular and coracoclavicular ligaments were then cut, and 1 of 6 reconstructions was performed: Weaver-Dunn, suture cerclage, and 4 different suture anchors. Acromioclavicular joint motion was reassessed, a cyclic loading test was performed, and the failure load was recorded.

Results: After Weaver-Dunn reconstruction, mean anteroposterior laxity increased from 8.8 ± 2.9 mm in the native state to 41.9 ± 7.6 mm (P ≤ .01), and mean superior laxity increased from 3.1 ± 1.5 mm to 13.6 ± 4.4 mm (P ≤ .01). Weaver-Dunn reconstructions failed at a lower load (177 ± 9 N) than all other reconstructions (range, 278–369 N) (P ≤ .05). Reconstruction using augmentative fixation allowed less acromioclavicular motion than Weaver-Dunn reconstruction (P ≤ .05) but more motion than the native ligaments (P ≤ .05). Specifically, mean superior laxity after reconstruction ranged between 6.5 and 9.0 mm compared with the native ligaments (3.1 ± 1.5 mm) and the Weaver-Dunn reconstructions (13.6 ± 4.4 mm). Mean anteroposterior laxity after the reconstructions tested ranged between 21.8 and 33.2 mm, compared with the native ligaments (8.8 ± 2.9 mm) and the Weaver-Dunn reconstructions (41.9 ± 7.6 mm).

Conclusion: Although none of the augmentative methods tested restored acromioclavicular stability to normal, all proved superior to the Weaver-Dunn reconstruction alone.

Clinical Relevance: This study suggests that when performing acromioclavicular reconstruction, supplemental fixation should be used because it provides more stability and pull-out strength than the Weaver-Dunn reconstruction alone.

Key Words: acromioclavicular • coracoclavicular • ligament • instability • suture anchor




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