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Musculoskeletal Research Center, Department of Orthopaedic Surgery, University of Pittsburgh, Pittsburgh, Pennsylvania
Presented at the 24th annual meeting of the AOSSM, Vancouver, British Columbia, Canada, July 1998.
Address correspondence and reprint requests to Christopher D. Harner, MD, Musculoskeletal Research Center, Department of Orthopaedic Surgery, University of Pittsburgh, POB 71199, Pittsburgh, PA 15213
We hypothesized that posterior cruciate ligament reconstructions are often compromised by associated injuries to the posterolateral structures. Therefore, we evaluated a posterior cruciate ligament reconstruction in isolated and combined injury models using a robotic/universal force-moment sensor testing system. The resulting knee kinematics and the in situ forces in the native and reconstructed posterior cruciate ligament were determined under four external loading conditions. In the isolated injury model, reconstruction reduced posterior tibial translation to within 1.5 ± 1.3 to 2.4 ± 1.4 mm of the intact knee at 30° and 90° under a 134-N posterior tibial load. In the combined injury model, deficiency of the posterolateral structures increased posterior tibial translation of the reconstructed knee by 6.0 ± 2.7 mm at 30° and 4.6 ± 1.5 mm at 90° of flexion. External rotation increased up to 14° while varus rotation increased up to 7°. In situ forces in the posterior cruciate ligament graft also increased significantly (by 22% to 150%) for all loading conditions. Our results demonstrate that a graft that restores knee kinematics for an isolated posterior cruciate ligament deficiency is rendered ineffective and may be overloaded if the posterolateral structures are deficient. Therefore, surgical reconstruction of both structures is recommended in the setting of a combined injury.
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