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The American Journal of Sports Medicine 26:389-394 (1998)
© 1998 American Orthopaedic Society for Sports Medicine

Isolated Sectioning of the Medial and Posteromedial Capsular Ligaments in the Posterior Cruciate Ligament-Deficient Knee

Influence on Posterior Tibial Translation

Joseph R. Ritchie, MD{dagger}, John A. Bergfeld, MD, Helen Kambic, MS and Timothy Manning

The Cleveland Clinic Foundation, Cleveland, Ohio

Presented at the interim meeting of the AOSSM, Atlanta, Georgia, February 1996.

{dagger} Address correspondence and reprint requests to Joseph R. Ritchie, MD, U.S. Center for Sports Medicine, 333 South Kirkwood Road, Suite 200, St. Louis, MO 63122

This study was undertaken to determine the contribution of various structures in the posterior cruciate ligament-deficient knee in resisting posterior tibial translation. With "isolated" injuries to the posterior cruciate ligament, the amount of posterior translation will decrease with the posterior drawer test as the knee is taken from neutral to internal tibial rotation. The present study was performed to conclusively determine the anatomic structure responsible for this clinical observation. The TestStar device was used to perform single-plane posterior drawer tests in 14 cadaveric knee specimens. The tests were performed with the knee in neutral tibial rotation and in 20° of internal tibial rotation. The intact knee was tested and then the knee was tested after sequential sectioning of the meniscofemoral ligaments, the posterior cruciate ligament, the posteromedial capsule, and the superficial medial collateral ligament. With the knee in neutral rotation, posterior translation continued to increase as each structure was sectioned. With the knee in internal tibial rotation, posterior displacement was significantly less than in neutral rotation for each state until the superficial medial collateral ligament was sectioned; posterior translation was increased after its sectioning. Our data demonstrate that the superficial medial collateral ligament is the structure responsible for a decrease in posterior tibial translation in the posterior cruciate ligament-deficient knee.




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